Preclinical-Stage Biotech • Marseille, France

Precision Medicine for
Hepatic Fibrosis

GenoTech Solutions develops genotype-guided antifibrotic therapies targeting the genetic drivers of liver fibrosis in MASH patients.

Explore Our Pipeline ↓
Breaking the Fibrosis Loop

GenoTech Solutions SAS is a preclinical-stage French biotechnology company developing a new class of genotype-guided antifibrotic therapies for metabolic dysfunction-associated steatohepatitis (MASH) with advanced hepatic fibrosis.

Our approach is built on twenty years of human genetic research demonstrating that germline variants in specific fibrogenic pathways act as master regulators of fibrosis severity across aetiologies and populations. These discoveries enable a precision medicine strategy where patients are stratified by genotype and treated with therapies matched to their molecular drivers.

Current approved MASH therapies achieve only 26–36% fibrosis improvement, leaving 64–74% of patients as non-responders. GenoTech addresses this unmet need with direct antifibrotic mechanisms and companion diagnostics to identify the patients most likely to benefit.

20+
Years of human genetic validation
3
Aetiologies confirmed
5
Populations studied
2
Therapeutic programmes
CDx
Companion diagnostics
SAS
Marseille, France
Dual-Pathway Antifibrotic Strategy
Two independent programmes targeting complementary fibrogenic pathways, designed for combination as the ultimate goal.
Drug Repositioning

GT-001

Repositioning of an approved drug with 15+ years of clinical safety data for a new antifibrotic indication in MASH. Genotype-guided patient selection via proprietary companion diagnostic.

FDA 505(b)(2) Phase 2a Ready CDx-Enabled
First-in-Class

GT-002

Discovery of a first-in-class small molecule inhibitor targeting a key protein–protein interaction in the fibrogenic transcriptional cascade. Inherent isoform selectivity validated computationally.

Fragment-to-Lead Computational + Biophysical Novel MoA
Combination

GT-001 + GT-002

Dual-pathway blockade interrupting the self-reinforcing fibrotic loop at both the transcriptional and hemodynamic nodes simultaneously. Expected superiority over monotherapy.

Synergistic MoA Non-Overlapping Safety Fixed-Dose Combination
Companion Diagnostic

Pharmacogenomic CDx

Proprietary SNP-based companion diagnostic identifying patients with genetically-driven rapid fibrosis progression. Enables precision enrichment of clinical trials and targeted prescribing.

Multi-Gene Panel 40–80% Prevalence IVD Development
From Human Genetics to Precision Therapeutics
Our programmes are anchored in two decades of genetic epidemiology across multiple populations and liver disease aetiologies.
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Genetic Causality

Germline variants in fibrogenic pathways identified as master regulators of fibrosis severity in cohorts spanning Chinese, Sudanese, Brazilian, Ugandan, and European populations. Odds ratios 1.5–4.0 across aetiologies.

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Computational Drug Design

Partnership with leading computational chemistry platforms for structure-based drug discovery. Homology modelling, pharmacophore generation, and virtual screening of >8 million compounds to identify selective hit molecules.

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Precision Enrichment

Companion diagnostic development enabling genotype-guided clinical trial enrichment. Standard of Care Plus design layered on existing metabolic therapies to address the residual fibrosis gap (64–74% non-responders).

Foundational Research
Key publications from the GenoTech scientific team on the genetics of hepatic fibrosis.
2020
Genetic algorithms identify individuals with high risk of severe liver disease caused by schistosomes
Dessein H et al. — Human Genetics, 139(6-7):821-831 • DOI: 10.1007/s00439-020-02160-4
2013
Genetic analysis of human predisposition to hepatosplenic disease: crucial role of connective tissue growth factor in rapid progression to severe hepatic fibrosis
Dessein A et al. — Hepatology, 57(3):1065-1075 • PMID: 23414795
2009
Variants of CTGF are associated with hepatic fibrosis in Chinese, Sudanese, and Brazilians infected with schistosomes
Dessein A et al. — Journal of Experimental Medicine, 206(11):2321-2328 • DOI: 10.1084/jem.20090383
2003
IFN-γ polymorphisms are associated with severe hepatic fibrosis in human hepatic schistosomiasis
Chevillard C, Dessein AJ et al. — Journal of Immunology, 171(10):5596-5601 • DOI: 10.4049/jimmunol.171.10.5596
1999
Severe hepatic fibrosis in S. mansoni infection is controlled by a major locus closely linked to the interferon-γ receptor gene
Dessein AJ et al. — American Journal of Human Genetics, 65(3):709-721 • DOI: 10.1086/302526
Founders & Scientific Team
Chief Executive Officer & Chief Technology Officer

Dr Laurent Gros, PhD

Co-founder. Background in pharmaceutical R&D, drug discovery programme management, translational medicine, and biotech operations. Leads scientific strategy, IP development, CRO partnerships, and investor relations. Responsible for the design and execution of both GT-001 and GT-002 programmes.

Co-Founder & Chief Scientific Officer

Prof. Alain Dessein, MD, PhD

25+ years of research in the genetics of hepatic fibrosis. Pioneered the identification of genetic determinants of fibrosis severity across multiple populations and aetiologies. Over 200 peer-reviewed publications. INSERM research director. Founder of the genetic epidemiology programme that underlies GenoTech’s therapeutic platform.

Partnership & Investor Enquiries

GenoTech Solutions is actively seeking strategic partnerships and Series A investment to advance its antifibrotic pipeline through clinical proof-of-concept.

laurent.gros@genotech.tech